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Friday, September 3, 2010
Aloe Vera Extract Heals Skin
Aloe Vera Extract Heals Skin
Aloe vera has been shown in clinical studies to have a positive effect on wound healing and holds anti-inflammatory properties.
As stretch marks start out as microscopic tears in skin layers due to overstretching of the dermis, aloe vera helps heal these small wounds without causing scar tissue to form, effectively preventing the appearance of stretch marks. Aloe vera has been used for thousands of years by the Egyptians, Romans, Greeks, Arabs, Indians and Chinese to decrease the appearance of scars and to moisturize and heal skin.
This extract from the aloe leaf protects skin from environmental factors and promotes skin regeneration.
For more information visit: http://www.revitol.com
Are There Alternative Therapies for Curing Hypothyroidism?
Are There Alternative Therapies For
Curing Hypothyroidism ?
In recent times, there has been so much focus on the effect of alternative remedies in treating ailments generally. Some even make claims that orthodox medicine cannot make. This has of course led to most people looking for alternative medicines and solutions to their ailments. Fortunately, some who have used these alternative remedies and have had good success with it. Hypothyroidism which is an illness stemming from the malfunction of the thyroid gland leading to the inadequate production of the thyroid gland’s hormone called thyroxine. The thyroid gland is found at the lower end of the neck just below the “Adam’s apple”. The thyroid gland like any well functioning organ does not just suddenly cease to function. Its malfunction is a product of certain unfavorable conditions. So, if certain steps are taken earlier, there won’t be any need in the first place to start treating hypothyroidism. There are some ways to effectively keep the thyroid gland in normal healthy condition.The very first move an individual can make is to be mindful of his diet. An individual’s diet is capable of either prolonging his existence on the surface of the earth, or helping him move faster to the grave than he ought to be moving. Watching what you eat is not only important in keeping away hypothyroidism, it is an effective way of keeping most diseases away. What you throw inside your belly is partially responsible for your general well being. So if you consume all that comes in your way without ay regards for your insides, then you might as well realize that your internal organs will be adversely affected rendering them ineffective. There are certain foods that will inhibit the production of thyroxine. Some of them are mustard greens, broccoli, soybeans, cauliflowers, cabbage et.cFoods like sugar, refined foods, dairy products should be avoided in excess. If you discover that you’ve got hypothyroidism, avoid caffeine, wheat and alcohol. Start including fatty acids in your diet. Taking about 1000-15000mg of these essential fatty acids thrice a day helps the thyroid glands in its production of thyroxine.Research has also shown that vitamins A, B complex, C and E are important in thyroxine production and in generally improving the thyroid’s health. Before taking tgem however, consult with your doctor as he would know in what quantities you should take them and for how long. Supplements like calcium, iodine L-tyrosine and selenium are also recommended by dieticians. Try all these, but ensure you run them by your doctor before you begin to use them.
For more information visit: http://www.thyromine.com
Antioxidant Levels Key to Prostate Cancer Risk in Some Men
Antioxidant levels key to prostate cancer risk in some men
Greater levels of selenium, vitamin E and the tomato carotenoid lycopene have been shown to reduce prostate cancer in one out of every four Caucasian males, or those who inherit a specific genetic variation that is particularly sensitive to oxidative stress, say US researchers.
Conversely, if carriers of this genetic variant have low levels of these vitamins and minerals, their risk of aggressive prostate increases substantially, as great as 10-fold, over those who maintain higher levels of these nutrients, they write in today’s issue of Cancer Research.
"This large prospective study provides further evidence that oxidative stress may be one of the important mechanisms for prostate cancer development and progression, and adequate intake of antioxidants, such as selenium, lycopene and vitamin E, may help prevent prostate cancer," said Dr Haojie Li, a researcher at the Brigham and Women's Hospital and Harvard Medical School.
The new findings are based on an analysis of 567 men diagnosed with prostate cancer between 1982 and 1995, and 764 cancer-free men from the Physicians Health Study.
The initial goal of this study was to assess the effect of aspirin and beta-carotene on men's health. Li’s team decided to check for variants of the gene that codes for manganese superoxide dismutatase (MnSOD), an important enzyme that works as an antioxidant in human cells to defend against disease.
The MnSOD gene is passed from parents to offspring in one of three forms: VV, VA or AA.
"Compared with men with the MnSOD VV or VA genotype, people with the AA genotype seem to be more sensitive to the antioxidant status," said Li. "Men with the AA genotype are more susceptible to prostate cancer if their antioxidant levels are low."
The study's results found that a quarter of the men in the study carried the MnSOD AA genotype, half carried the VA genotype, and the remaining quarter carried the VV genotype.
The results indicated that the VA and VV men were at equivalent risk for developing prostate cancer across all levels of antioxidants in their blood.
But compared to MnSOD VV or VA carriers in the lowest quartile of selenium levels, MnSOD AA males had an 89 per cent greater risk for developing aggressive prostate cancer if they had low blood levels of the mineral.
On the other hand, MnSOD AA carriers with high selenium – those men in the highest quartile – had a 65 per cent lower risk than the MnSOD VV or VA males who maintained low levels of selenium.
"The levels of selenium in the highest quartile of these men are not abnormally high," Li said. "Our range is neither extremely high nor extremely low."
While similar trends were observed for lycopene and vitamin E when tested independently, the contrast in relative risk was most pronounced for the men who had high blood levels for all three antioxidants combined, said the researchers.
"Among men with the MnSOD AA genotype, we observed a 10-fold difference in risk for aggressive prostate cancer, when comparing men with high versus low levels of antioxidants combined,"said Li. "In contrast, among men with the VV or VA genotype, the prostate cancer risk was only weakly altered by these antioxidant levels."
"Our study, as well as many other epidemiological studies, encourages dietary intake of nutrients such as lycopene from tomato products, or supplements for vitamin E and selenium to reduce risk of prostate cancer," said Li.
Prostate cancer is one of the biggest cancer killers in industrial countries and affects more than 500,000 men worldwide every year. This number is expected to increase with the ageing population.
Similar interactions between dietary antioxidants and the variations in the MnSOD gene have previously been linked to risk for breast cancer.
For more information visit: http://www.prostacet.com
Cause of irritable bowel syndrome
Irritable bowel syndrome is
believed to be due to the abnormal function (dysfunction) of the
muscles of the organs of the gastrointestinal tract or the nerves
controlling the organs. The nervous control of the gastrointestinal
tract, however, is complex. A system of nerves runs the entire length
of the gastrointestinal tract from the esophagus to the anus in the
muscular walls of the organs. These nerves communicate with other
nerves that travel to and from the spinal cord. Nerves within the
spinal cord, in turn, travel to and from the brain. (The
gastrointestinal tract is exceeded in the numbers of nerves it
contains only by the spinal cord and brain.) Thus, the abnormal
function of the nervous system in IBS may occur in a gastrointestinal
muscular organ, the spinal cord, or the brain.
The nervous system that controls the gastrointestinal organs, as with
most other organs, contains both sensory and motor nerves. The sensory
nerves continuously sense what is happening within the organ and relay
this information to nerves in the organ's wall. From there,
information can be relayed to the spinal cord and brain. The
information is received and processed in the organ's wall, the spinal
cord, or the brain. Then, based on this sensory input and the way the
input is processed, commands (responses) are sent to the organ over
the motor nerves. Two of the most common motor responses in the
intestine are contraction or relaxation of the muscle of the organ and
secretion of fluid and/or mucus into the organ.
As already mentioned, abnormal function of the nerves of the
gastrointestinal organs, at least theoretically, might occur in the
organ, spinal cord, or brain. Moreover, the abnormalities might occur
in the sensory nerves, the motor nerves, or at processing centers in
the intestine, spinal cord, or brain. Some researchers argue that the
cause of functional diseases is abnormalities in the function of the
sensory nerves. For example, normal activities, such as stretching of
the small intestine by food, may give rise to abnormal sensory signals
that are sent to the spinal cord and brain, where they are perceived
as pain.
Other researchers argue that the cause of functional
diseases is abnormalities in the function of the motor nerves. For
example, abnormal commands through the motor nerves might produce a
painful spasm (contraction) of the muscles. Still others argue that
abnormally functioning processing centers are responsible for
functional diseases because they misinterpret normal sensations or
send abnormal commands to the organ. In fact, some functional diseases
may be due to sensory dysfunction, motor dysfunction, or both sensory
and motor dysfunction. Still others may be due to abnormalities within
the processing centers One area that is receiving a great deal of
scientific attention is the potential role of gas produced by
intestinal bacteria in patients with IBS. Studies have demonstrated
that patients with IBS produce larger amounts of gas than individuals
without IBS, and the gas may be retained longer in the small
intestine. Among patients with IBS, abdominal size increases over the
day, reaching a maximum in the evening and returning to baseline by
the following morning. In individuals without IBS, there is no
increase in abdominal size during the day.
There has been a great deal of controversy over the role that poor
digestion and/or absorption of dietary sugars may play in aggravating
the symptoms of IBS. Poor digestion of lactose, the sugar in milk, is
very common as is poor absorption of fructose, a sweetener found in
many processed foods. Poor digestion or absorption of these sugars
could aggravate the symptoms of IBS since unabsorbed sugars often
cause increased formation of gas.
Although these abnormalities in production and transport of gas could
give rise to some of the symptoms of IBS, much more work will need to
be done before the role of intestinal gas in IBS is clear.
Dietary fat in healthy individuals causes food as well as gas to move
more slowly through the stomach and small intestine. Some patients with
IBS may even respond to dietary fat in an exaggerated fashion with
greater slowing. Thus, dietary fat could--and probably does--aggravate
the symptoms of IBS.
believed to be due to the abnormal function (dysfunction) of the
muscles of the organs of the gastrointestinal tract or the nerves
controlling the organs. The nervous control of the gastrointestinal
tract, however, is complex. A system of nerves runs the entire length
of the gastrointestinal tract from the esophagus to the anus in the
muscular walls of the organs. These nerves communicate with other
nerves that travel to and from the spinal cord. Nerves within the
spinal cord, in turn, travel to and from the brain. (The
gastrointestinal tract is exceeded in the numbers of nerves it
contains only by the spinal cord and brain.) Thus, the abnormal
function of the nervous system in IBS may occur in a gastrointestinal
muscular organ, the spinal cord, or the brain.
The nervous system that controls the gastrointestinal organs, as with
most other organs, contains both sensory and motor nerves. The sensory
nerves continuously sense what is happening within the organ and relay
this information to nerves in the organ's wall. From there,
information can be relayed to the spinal cord and brain. The
information is received and processed in the organ's wall, the spinal
cord, or the brain. Then, based on this sensory input and the way the
input is processed, commands (responses) are sent to the organ over
the motor nerves. Two of the most common motor responses in the
intestine are contraction or relaxation of the muscle of the organ and
secretion of fluid and/or mucus into the organ.
As already mentioned, abnormal function of the nerves of the
gastrointestinal organs, at least theoretically, might occur in the
organ, spinal cord, or brain. Moreover, the abnormalities might occur
in the sensory nerves, the motor nerves, or at processing centers in
the intestine, spinal cord, or brain. Some researchers argue that the
cause of functional diseases is abnormalities in the function of the
sensory nerves. For example, normal activities, such as stretching of
the small intestine by food, may give rise to abnormal sensory signals
that are sent to the spinal cord and brain, where they are perceived
as pain.
Other researchers argue that the cause of functional
diseases is abnormalities in the function of the motor nerves. For
example, abnormal commands through the motor nerves might produce a
painful spasm (contraction) of the muscles. Still others argue that
abnormally functioning processing centers are responsible for
functional diseases because they misinterpret normal sensations or
send abnormal commands to the organ. In fact, some functional diseases
may be due to sensory dysfunction, motor dysfunction, or both sensory
and motor dysfunction. Still others may be due to abnormalities within
the processing centers One area that is receiving a great deal of
scientific attention is the potential role of gas produced by
intestinal bacteria in patients with IBS. Studies have demonstrated
that patients with IBS produce larger amounts of gas than individuals
without IBS, and the gas may be retained longer in the small
intestine. Among patients with IBS, abdominal size increases over the
day, reaching a maximum in the evening and returning to baseline by
the following morning. In individuals without IBS, there is no
increase in abdominal size during the day.
There has been a great deal of controversy over the role that poor
digestion and/or absorption of dietary sugars may play in aggravating
the symptoms of IBS. Poor digestion of lactose, the sugar in milk, is
very common as is poor absorption of fructose, a sweetener found in
many processed foods. Poor digestion or absorption of these sugars
could aggravate the symptoms of IBS since unabsorbed sugars often
cause increased formation of gas.
Although these abnormalities in production and transport of gas could
give rise to some of the symptoms of IBS, much more work will need to
be done before the role of intestinal gas in IBS is clear.
Dietary fat in healthy individuals causes food as well as gas to move
more slowly through the stomach and small intestine. Some patients with
IBS may even respond to dietary fat in an exaggerated fashion with
greater slowing. Thus, dietary fat could--and probably does--aggravate
the symptoms of IBS.
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